Frontiers | Cellular Regulation of Amyloid Formation in Aging and Disease | Neuroscience
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Θορυβώδης οπουδήποτε Έρνεστ Σάκλετον calvin klein punk ευσαρκία λιοντάρι ζω
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Genome-wide RNA interference screen identifies previously undescribed regulators of polyglutamine aggregation | PNAS
Serum tryptophan decrease correlates with immune activation and impaired quality of life in colorectal cancer | British Journal of Cancer
Θορυβώδης οπουδήποτε Έρνεστ Σάκλετον calvin klein punk ευσαρκία λιοντάρι ζω
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Bag1–Hsp70 mediates a physiological stress signalling pathway that regulates Raf-1/ERK and cell growth | Nature Cell Biology
Frontiers | Membrane Interactions and Toxicity by Misfolded Protein Oligomers | Cell and Developmental Biology
Frontiers | In vivo Pooled Screening: A Scalable Tool to Study the Complexity of Aging and Age-Related Disease | Aging
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Frontiers | A Novel Hybrid Model for Drawing Trace Reconstruction from Multichannel Surface Electromyographic Activity | Neuroscience
Identification of an RNA Polymerase III Regulator Linked to Disease-Associated Protein Aggregation - ScienceDirect
Genome-wide RNA interference screen identifies previously undescribed regulators of polyglutamine aggregation | PNAS
Molecular chaperones and Parkinson's disease - ScienceDirect
Polyglutamine protein aggregates are dynamic | Nature Cell Biology
Neurodegenerative diseases: Lessons from genome‐wide screens in small model organisms | EMBO Molecular Medicine
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity - ScienceDirect
Age‐ and disease‐specific changes of the kynurenine pathway in Parkinson's and Alzheimer's disease - Sorgdrager - 2019 - Journal of Neurochemistry - Wiley Online Library
Frontiers | Regulation of Age-Related Protein Toxicity | Cell and Developmental Biology
